Choking the Feeder
نویسنده
چکیده
Platelet activity usually inversely correlates with serum albumin levels (e.g., in course of remission or relapse), and it is potentially reversible by adding albumin in vitro or following albumin infusions. Furthermore, many other important hemostatic proteins of similar size are also pathologically excreted in the urine. The loss of antithrombotic factors in the urine is counterbalanced by an increase in plasma, procoagulant cofactors, such as factor V, factor VIII, and fibrinogen.[4]
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